[真男同性恋的发病机制(雄激素功能不全?)]。

Revista iberica de endocrinologia Pub Date : 1976-05-01
F A de la Balze
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引用次数: 0

摘要

临床和实验研究表明,在胚胎发育或围产期,雄激素不足可能是同性恋行为异常发病机制的决定性因素。作者认为,在真正的男同性恋者身上看到的雄激素异常没有致病意义,而是围产期胚胎发生期间发生的重复,当时这些异常具有致病作用。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
[Etiopathogenesis of true male homosexuality (androgenic insufficiency?)].

Clinical and experimental studies have suggested the author the possibility that an androgenic insufficiency, during embryogenesis or the perinatal period, may be a determinant factor in the pathogenesis of the anomalies of homosexual behavior. In the author's opinion, the androgenic anomalies seen in the genuine male homosexual have no pathogenic significance, but are a repetition of what occured during embryogenesis of the perinatal period, at the time when these anomalies had a pathogenic role.

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