U. Park, Hyun Tae Lee, Yujin Kim, T. Jeong, Nahyun Gu, Y. Heo
{"title":"burrosumab Fab片段的晶体结构,burrosumab是治疗x连锁低磷血症和肿瘤性骨软化症的一流抗fgf23药物","authors":"U. Park, Hyun Tae Lee, Yujin Kim, T. Jeong, Nahyun Gu, Y. Heo","doi":"10.34184/kssb.2021.9.4.59","DOIUrl":null,"url":null,"abstract":"","PeriodicalId":153406,"journal":{"name":"BIODESIGN","volume":"255 1","pages":"0"},"PeriodicalIF":0.0000,"publicationDate":"2021-12-30","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":"0","resultStr":"{\"title\":\"Crystal structure of the Fab fragment of burosumab, the first-in-class anti-FGF23 medicine for treating X-linked hypophosphatemia and tumor-induced osteomalacia\",\"authors\":\"U. Park, Hyun Tae Lee, Yujin Kim, T. Jeong, Nahyun Gu, Y. Heo\",\"doi\":\"10.34184/kssb.2021.9.4.59\",\"DOIUrl\":null,\"url\":null,\"abstract\":\"\",\"PeriodicalId\":153406,\"journal\":{\"name\":\"BIODESIGN\",\"volume\":\"255 1\",\"pages\":\"0\"},\"PeriodicalIF\":0.0000,\"publicationDate\":\"2021-12-30\",\"publicationTypes\":\"Journal Article\",\"fieldsOfStudy\":null,\"isOpenAccess\":false,\"openAccessPdf\":\"\",\"citationCount\":\"0\",\"resultStr\":null,\"platform\":\"Semanticscholar\",\"paperid\":null,\"PeriodicalName\":\"BIODESIGN\",\"FirstCategoryId\":\"1085\",\"ListUrlMain\":\"https://doi.org/10.34184/kssb.2021.9.4.59\",\"RegionNum\":0,\"RegionCategory\":null,\"ArticlePicture\":[],\"TitleCN\":null,\"AbstractTextCN\":null,\"PMCID\":null,\"EPubDate\":\"\",\"PubModel\":\"\",\"JCR\":\"\",\"JCRName\":\"\",\"Score\":null,\"Total\":0}","platform":"Semanticscholar","paperid":null,"PeriodicalName":"BIODESIGN","FirstCategoryId":"1085","ListUrlMain":"https://doi.org/10.34184/kssb.2021.9.4.59","RegionNum":0,"RegionCategory":null,"ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":null,"EPubDate":"","PubModel":"","JCR":"","JCRName":"","Score":null,"Total":0}
Crystal structure of the Fab fragment of burosumab, the first-in-class anti-FGF23 medicine for treating X-linked hypophosphatemia and tumor-induced osteomalacia