牛津-阿斯利康COVID-19疫苗接种后全身性药物相关的三叶间性和弯曲性皮疹样皮疹。

Q2 Medicine
Danilo Di Bona, Andrea Miniello, Eustachio Nettis
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引用次数: 1

摘要

全身性药物相关性三叉戟和弯曲性皮疹(SDRIFE)是一种药物不良反应,表现为全身药物暴露后皮肤褶皱的对称红斑皮疹。自COVID-19大流行开始以来,文献中已经报道了与使用不同的抗sars - cov -2疫苗相关的大量可能的副作用,但在这种情况下,只有少数几次描述了类似sdrif的爆发。我们在这里讨论一个病例的sdrif样爆发后,第二剂牛津-阿斯利康Vaxzevria疫苗。
本文章由计算机程序翻译,如有差异,请以英文原文为准。

Systemic drug-related intertriginous and flexural exanthema-like eruption after Oxford-AstraZeneca COVID-19 vaccine.

Systemic drug-related intertriginous and flexural exanthema-like eruption after Oxford-AstraZeneca COVID-19 vaccine.

Systemic drug-related intertriginous and flexural exanthema-like eruption after Oxford-AstraZeneca COVID-19 vaccine.

Systemic drug-related intertriginous and flexural exanthema (SDRIFE) is an adverse drug reaction which manifests as a symmetrical erythematous rash involving the skin folds after systemic drug exposure. A vast array of possible side effects associated with administration of different anti-SARS-CoV-2 vaccines have been reported in literature since the beginning of the COVID-19 pandemic, but only few times SDRIFE-like eruptions have been described in this context. We discuss here a case of SDRIFE-like eruption following the second dose of Oxford-Astrazeneca Vaxzevria vaccine.

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来源期刊
Clinical and Molecular Allergy
Clinical and Molecular Allergy Medicine-Immunology and Allergy
CiteScore
8.20
自引率
0.00%
发文量
11
审稿时长
13 weeks
期刊介绍: Clinical and Molecular Allergy is an open access, peer-reviewed, online journal that publishes research on human allergic and immunodeficient disease (immune deficiency not related to HIV infection/AIDS). The scope of the journal encompasses all aspects of the clinical, genetic, molecular and inflammatory aspects of allergic-respiratory (Type 1 hypersensitivity) and non-AIDS immunodeficiency disorders. However, studies of allergic/hypersensitive aspects of HIV infection/AIDS or drug desensitization protocols in AIDS are acceptable. At the basic science level, this includes original work and reviews on the genetic and molecular mechanisms underlying the inflammatory response.
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