退出通知:CircRNA circPRKCI在骨质疏松和海绵miR-103中上调抑制脂多糖(LPS)诱导的成骨细胞凋亡

IF 2 4区 医学 Q3 ENDOCRINOLOGY & METABOLISM
Yue Liu, Hongchao Huang, Jianan Li, Kaihui Zhang, Hongfeng Jiang
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引用次数: 0

摘要

由于作者没有回应编辑的要求来满足编辑的要求,因此,文章被撤回。边沁科学为由此造成的不便向本刊读者道歉。边沁文章撤回编辑政策可在https://benthamscience.com/editorial-policies-main.php.Bentham科学免责声明中找到:投稿本期刊的稿件未发表,不会同时投稿或在其他地方发表,这是发表的条件。此外,在其他地方发表的任何数据、插图、结构或表格必须报告,并必须获得版权许可才能复制。抄袭是严格禁止的,通过提交文章发表,作者同意出版商有法律权利对作者采取适当的行动,如果发现抄袭或捏造信息。通过提交手稿,作者同意如果文章被接受出版,其文章的版权将转移给出版商。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
Withdrawal Notice: CircRNA circPRKCI is upregulated in osteoporosis and sponges miR-103 to suppress the apoptosis of osteoblast induced by Lipopolysaccharide (LPS)

Since the authors are not responding to the editor’s requests to fulfill the editorial requirement, therefore, the article has been withdrawn.

Bentham Science apologizes to the readers of the journal for any inconvenience this may have caused.

The Bentham Editorial Policy on Article Withdrawal can be found at https://benthamscience.com/editorial-policies-main.php.

Bentham science disclaimer: It is a condition of publication that manuscripts submitted to this journal have not been published and will not be simultaneously submitted or published elsewhere. Furthermore, any data, illustration, structure or table that has been published elsewhere must be reported, and copyright permission for reproduction must be obtained. Plagiarism is strictly forbidden, and by submitting the article for publication the authors agree that the publishers have the legal right to take appropriate action against the authors, if plagiarism or fabricated information is discovered. By submitting a manuscript the authors agree that the copyright of their article is transferred to the publishers if and when the article is accepted for publication.

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来源期刊
Endocrine, metabolic & immune disorders drug targets
Endocrine, metabolic & immune disorders drug targets ENDOCRINOLOGY & METABOLISMIMMUNOLOGY-IMMUNOLOGY
CiteScore
4.60
自引率
5.30%
发文量
217
期刊介绍: Aims & Scope This journal is devoted to timely reviews and original articles of experimental and clinical studies in the field of endocrine, metabolic, and immune disorders. Specific emphasis is placed on humoral and cellular targets for natural, synthetic, and genetically engineered drugs that enhance or impair endocrine, metabolic, and immune parameters and functions. Moreover, the topics related to effects of food components and/or nutraceuticals on the endocrine-metabolic-immune axis and on microbioma composition are welcome.
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