ARID5A promotes inflammation and fibrosis during cardiac aging

IF 10.8 Q1 CARDIAC & CARDIOVASCULAR SYSTEMS
Shoval Miyara, Eldad Tzahor
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引用次数: 0

Abstract

A systematic biology approach identifies an RNA-binding protein, ARID5A, as a regulator of inflammation and fibrosis in the aging human heart. Inhibition of ARID5A reduced inflammation and fibrosis and improved cardiac function in old mice.

Abstract Image

ARID5A在心脏老化过程中促进炎症和纤维化。
一种系统的生物学方法确定了一种rna结合蛋白ARID5A,作为衰老人类心脏炎症和纤维化的调节因子。抑制ARID5A可减轻老年小鼠的炎症和纤维化,改善心功能。
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CiteScore
5.70
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