RETRACTION: CDKL5 Promotes Proliferation, Migration, and Chemotherapeutic Drug Resistance of Glioma Cells via Activation of the PI3K/AKT Signaling Pathway.

IF 2.8 4区 生物学 Q3 BIOCHEMISTRY & MOLECULAR BIOLOGY
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引用次数: 0

Abstract

Retraction: Z. Jiang, T. Gong, and H. Wei, "CDKL5 Promotes Proliferation, Migration, and Chemotherapeutic Drug Resistance of Glioma Cells via Activation of the PI3K/AKT Signaling Pathway," FEBS Open Bio 10, no. 2 (2020): 268-277, https://doi.org/10.1002/2211-5463.12780. The above article, published online on 21 January 2020, in Wiley Online Library (wileyonlinelibrary.com), has been retracted by agreement between the journal Editor-in-Chief, Miguel A. De la Rosa; FEBS Press; and John Wiley and Sons Ltd. The retraction has been agreed upon following an investigation into concerns raised by a third party, which revealed inappropriate image duplications between this (Figure 3E, 4A and E, 5B and E) and other articles that were previously published or published in the same or following year. Given the extent of the identified issues, the editors have lost confidence in the data presented and consider the conclusions of this manuscript substantially compromised.

回顾:CDKL5通过激活PI3K/AKT信号通路促进胶质瘤细胞的增殖、迁移和化疗耐药。
引用本文:蒋振华,龚涛,魏辉,“CDKL5在神经胶质瘤细胞增殖、迁移和化疗耐药中的作用:PI3K/AKT信号通路的激活,”中国生物医学工程杂志,第10期。2 (2020): 268-277, https://doi.org/10.1002/2211-5463.12780。上述文章于2020年1月21日在线发表在Wiley在线图书馆(wileyonlinelibrary.com)上,经该杂志主编Miguel A. De la Rosa;2月出版社;及约翰威利父子有限公司。在对第三方提出的问题进行调查后,我们同意撤回这篇文章(图3E、图4A和图E、图5B和图E)与之前发表或同年或次年发表的其他文章之间存在不适当的图像重复。鉴于已确定问题的程度,编辑对所提供的数据失去了信心,并认为这篇手稿的结论在很大程度上受到了损害。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
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来源期刊
FEBS Open Bio
FEBS Open Bio BIOCHEMISTRY & MOLECULAR BIOLOGY-
CiteScore
5.10
自引率
0.00%
发文量
173
审稿时长
10 weeks
期刊介绍: FEBS Open Bio is an online-only open access journal for the rapid publication of research articles in molecular and cellular life sciences in both health and disease. The journal''s peer review process focuses on the technical soundness of papers, leaving the assessment of their impact and importance to the scientific community. FEBS Open Bio is owned by the Federation of European Biochemical Societies (FEBS), a not-for-profit organization, and is published on behalf of FEBS by FEBS Press and Wiley. Any income from the journal will be used to support scientists through fellowships, courses, travel grants, prizes and other FEBS initiatives.
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