Correction: Endometrial senescence is mediated by interleukin 17 receptor B signaling

IF 8.2 2区 生物学 Q1 CELL BIOLOGY
Keiko Kawamura, Yumiko Matsumura, Teruhiko Kawamura, Hiromitsu Araki, Norio Hamada, Kazutaka Kuramoto, Hiroshi Yagi, Ichiro Onoyama, Kazuo Asanoma, Kiyoko Kato
{"title":"Correction: Endometrial senescence is mediated by interleukin 17 receptor B signaling","authors":"Keiko Kawamura, Yumiko Matsumura, Teruhiko Kawamura, Hiromitsu Araki, Norio Hamada, Kazutaka Kuramoto, Hiroshi Yagi, Ichiro Onoyama, Kazuo Asanoma, Kiyoko Kato","doi":"10.1186/s12964-024-01754-z","DOIUrl":null,"url":null,"abstract":"<p><b>Correction: Cell Commun Signal 22, 363 (2024)</b>.</p><p><b>https://doi.org/10.1186/s12964-024-01740-5</b>.</p><p> Following publication of the original article [1], the authors reported that the incorrect additional file 12 was published. The correct additional file is published in this correction article and the original article [1] has been corrected.</p><ul data-track-component=\"outbound reference\" data-track-context=\"references section\"><li><p>Kawamura, K., Matsumura, Y., Kawamura, T. et al. Endometrial senescence is mediated by interleukin 17 receptor B signaling. <i>Cell Commun Signal</i><b>22</b>, 363 (2024). https://doi.org/10.1186/s12964-024-01740-5</p></li></ul><p>Download references<svg aria-hidden=\"true\" focusable=\"false\" height=\"16\" role=\"img\" width=\"16\"><use xlink:href=\"#icon-eds-i-download-medium\" xmlns:xlink=\"http://www.w3.org/1999/xlink\"></use></svg></p><h3>Authors and Affiliations</h3><ol><li><p>Department of Obstetrics and Gynecology, Graduate School of Medical Sciences, Kyushu University, Maidashi 3-1-1, Higashi-ku, Fukuoka, 812-8582, Japan</p><p>Keiko Kawamura, Yumiko Matsumura, Teruhiko Kawamura, Norio Hamada, Kazutaka Kuramoto, Hiroshi Yagi, Ichiro Onoyama, Kazuo Asanoma &amp; Kiyoko Kato</p></li><li><p>Department of Business and Technology Management, Faculty of Economics, Kyushu University, Fukuoka, Japan</p><p>Hiromitsu Araki</p></li></ol><span>Authors</span><ol><li><span>Keiko Kawamura</span>View author publications<p>You can also search for this author in <span>PubMed<span> </span>Google Scholar</span></p></li><li><span>Yumiko Matsumura</span>View author publications<p>You can also search for this author in <span>PubMed<span> </span>Google Scholar</span></p></li><li><span>Teruhiko Kawamura</span>View author publications<p>You can also search for this author in <span>PubMed<span> </span>Google Scholar</span></p></li><li><span>Hiromitsu Araki</span>View author publications<p>You can also search for this author in <span>PubMed<span> </span>Google Scholar</span></p></li><li><span>Norio Hamada</span>View author publications<p>You can also search for this author in <span>PubMed<span> </span>Google Scholar</span></p></li><li><span>Kazutaka Kuramoto</span>View author publications<p>You can also search for this author in <span>PubMed<span> </span>Google Scholar</span></p></li><li><span>Hiroshi Yagi</span>View author publications<p>You can also search for this author in <span>PubMed<span> </span>Google Scholar</span></p></li><li><span>Ichiro Onoyama</span>View author publications<p>You can also search for this author in <span>PubMed<span> </span>Google Scholar</span></p></li><li><span>Kazuo Asanoma</span>View author publications<p>You can also search for this author in <span>PubMed<span> </span>Google Scholar</span></p></li><li><span>Kiyoko Kato</span>View author publications<p>You can also search for this author in <span>PubMed<span> </span>Google Scholar</span></p></li></ol><h3>Corresponding author</h3><p>Correspondence to Kiyoko Kato.</p><h3>Publisher’s Note</h3><p>Springer Nature remains neutral with regard to jurisdictional claims in published maps and institutional affiliations.</p><p>The online version of the original article can be found at https://doi.org/10.1186/s12964-024-01740-5.</p><p>Below is the link to the electronic supplementary material.</p><h3>12964_2024_1754_MOESM1_ESM.mp4</h3><p> Additional file 12: Video 3. The first seven days time-lapse videos showing organoid forming of IL17RB(-) subpopulations in the presence of either a JNK inhibitor only (SP600125; 5 µM), IL17B only (100 ng/ml), both, or neither (control).</p><p><b>Open Access</b> This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. 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Correction: Endometrial senescence is mediated by interleukin 17 receptor B signaling. <i>Cell Commun Signal</i> <b>22</b>, 370 (2024). https://doi.org/10.1186/s12964-024-01754-z</p><p>Download citation<svg aria-hidden=\"true\" focusable=\"false\" height=\"16\" role=\"img\" width=\"16\"><use xlink:href=\"#icon-eds-i-download-medium\" xmlns:xlink=\"http://www.w3.org/1999/xlink\"></use></svg></p><ul data-test=\"publication-history\"><li><p>Published<span>: </span><span><time datetime=\"2024-07-22\">22 July 2024</time></span></p></li><li><p>DOI</abbr><span>: </span><span>https://doi.org/10.1186/s12964-024-01754-z</span></p></li></ul><h3>Share this article</h3><p>Anyone you share the following link with will be able to read this content:</p><button data-track=\"click\" data-track-action=\"get shareable link\" data-track-external=\"\" data-track-label=\"button\" type=\"button\">Get shareable link</button><p>Sorry, a shareable link is not currently available for this article.</p><p 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Abstract

Correction: Cell Commun Signal 22, 363 (2024).

https://doi.org/10.1186/s12964-024-01740-5.

Following publication of the original article [1], the authors reported that the incorrect additional file 12 was published. The correct additional file is published in this correction article and the original article [1] has been corrected.

  • Kawamura, K., Matsumura, Y., Kawamura, T. et al. Endometrial senescence is mediated by interleukin 17 receptor B signaling. Cell Commun Signal22, 363 (2024). https://doi.org/10.1186/s12964-024-01740-5

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Authors and Affiliations

  1. Department of Obstetrics and Gynecology, Graduate School of Medical Sciences, Kyushu University, Maidashi 3-1-1, Higashi-ku, Fukuoka, 812-8582, Japan

    Keiko Kawamura, Yumiko Matsumura, Teruhiko Kawamura, Norio Hamada, Kazutaka Kuramoto, Hiroshi Yagi, Ichiro Onoyama, Kazuo Asanoma & Kiyoko Kato

  2. Department of Business and Technology Management, Faculty of Economics, Kyushu University, Fukuoka, Japan

    Hiromitsu Araki

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Correspondence to Kiyoko Kato.

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The online version of the original article can be found at https://doi.org/10.1186/s12964-024-01740-5.

Below is the link to the electronic supplementary material.

12964_2024_1754_MOESM1_ESM.mp4

Additional file 12: Video 3. The first seven days time-lapse videos showing organoid forming of IL17RB(-) subpopulations in the presence of either a JNK inhibitor only (SP600125; 5 µM), IL17B only (100 ng/ml), both, or neither (control).

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Kawamura, K., Matsumura, Y., Kawamura, T. et al. Correction: Endometrial senescence is mediated by interleukin 17 receptor B signaling. Cell Commun Signal 22, 370 (2024). https://doi.org/10.1186/s12964-024-01754-z

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更正:子宫内膜衰老由白细胞介素 17 受体 B 信号传导介导
更正:Cell Commun Signal 22, 363 (2024).https://doi.org/10.1186/s12964-024-01740-5.原文[1]发表后,作者报告说发表了错误的附加文件12。Kawamura, K., Matsumura, Y., Kawamura, T. et al. Endometrial senescence is mediated by interleukin 17 receptor B signaling.Cell Commun Signal22, 363 (2024). https://doi.org/10.1186/s12964-024-01740-5下载参考文献作者及单位九州大学医学部研究生院妇产科,Maidashi 3-1-1, Higashi-ku, Fukuoka, 812-8582, JapanKeiko Kawamura, Yumiko Matsumura, Teruhiko Kawamura, Norio Hamada, Kazutaka Kuramoto, Hiroshi Yagi, Ichiro Onoyama, Kazuo Asanoma &;Kiyoko Kato九州大学经济学部商业与技术管理系,福冈、日本Hiromitsu Araki作者Keiko Kawamura查看作者发表的论文您也可以在PubMed Google Scholar中搜索该作者Yumiko Matsumura查看作者发表的论文您也可以在PubMed Google Scholar中搜索该作者Teruhiko Kawamura查看作者发表的论文您也可以在PubMed Google Scholar中搜索该作者您也可以在 PubMed Google ScholarHiromitsu ArakiView 作者发表的作品您也可以在 PubMed Google ScholarNorio HamadaView 作者发表的作品您也可以在 PubMed Google ScholarKazutakaKuramotoView 作者发表作品您也可以在 PubMed Google Scholar中搜索该作者Hiroshi YagiView 作者发表作品您也可以在 PubMed Google Scholar中搜索该作者Ichiro OnoyamaView 作者发表作品您也可以在 PubMed Google Scholar中搜索该作者Kazuo AsanomaView 作者发表作品您也可以在 PubMed Google Scholar中搜索该作者Kiyoko KatoView 作者发表作品您也可以在 PubMed Google Scholar中搜索该作者通讯作者Kiyoko Kato.出版商注释施普林格-自然(Springer Nature)对出版地图中的管辖权主张和机构隶属关系保持中立。原文的在线版本可在 https://doi.org/10.1186/s12964-024-01740-5.Below 找到,这是电子补充材料的链接。12964_2024_1754_MOESM1_ESM.mp4 附加文件 12:视频 3。前七天的延时视频显示了在仅使用 JNK 抑制剂(SP600125;5 µM)、仅使用 IL17B(100 ng/ml)、两者都使用或两者都不使用(对照)的情况下 IL17RB(-)亚群形成的类器官。0 国际许可协议,该协议允许以任何媒介或格式使用、共享、改编、分发和复制本文,但须注明原作者和出处,提供知识共享许可协议的链接,并说明是否进行了修改。本文中的图片或其他第三方材料均包含在文章的知识共享许可协议中,除非在材料的署名栏中另有说明。如果材料未包含在文章的知识共享许可协议中,且您打算使用的材料不符合法律规定或超出许可使用范围,您需要直接从版权所有者处获得许可。要查看该许可的副本,请访问 http://creativecommons.org/licenses/by/4.0/。除非在数据的信用行中另有说明,否则知识共享公共领域专用免责声明 (http://creativecommons.org/publicdomain/zero/1.0/) 适用于本文提供的数据。转载与许可引用本文Kawamura, K., Matsumura, Y., Kawamura, T. et al. Correction:子宫内膜衰老由白细胞介素 17 受体 B 信号传导介导。Cell Commun Signal 22, 370 (2024). https://doi.org/10.1186/s12964-024-01754-zDownload citationPublished: 22 July 2024DOI: https://doi.org/10.1186/s12964-024-01754-zShare this articleAnyone you share the following link with will be able to read this content:Get shareable linkSorry, a shareable link is not currently available for this article.Copy to clipboard Provided by the Springer Nature SharedIt content-sharing initiative
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来源期刊
CiteScore
11.00
自引率
0.00%
发文量
180
期刊介绍: Cell Communication and Signaling (CCS) is a peer-reviewed, open-access scientific journal that focuses on cellular signaling pathways in both normal and pathological conditions. It publishes original research, reviews, and commentaries, welcoming studies that utilize molecular, morphological, biochemical, structural, and cell biology approaches. CCS also encourages interdisciplinary work and innovative models, including in silico, in vitro, and in vivo approaches, to facilitate investigations of cell signaling pathways, networks, and behavior. Starting from January 2019, CCS is proud to announce its affiliation with the International Cell Death Society. The journal now encourages submissions covering all aspects of cell death, including apoptotic and non-apoptotic mechanisms, cell death in model systems, autophagy, clearance of dying cells, and the immunological and pathological consequences of dying cells in the tissue microenvironment.
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