Publisher Correction: E3 ubiquitin ligase UBR5 modulates circadian rhythm by facilitating the ubiquitination and degradation of the key clock transcription factor BMAL1.

IF 6.9 1区医学 Q1 CHEMISTRY, MULTIDISCIPLINARY

Acta Pharmacologica Sinica Pub Date : 2024-12-01 DOI:10.1038/s41401-024-01328-2

Chun-Yan Duan, Yue Li, Hao-Yu Zhi, Yao Tian, Zheng-Yun Huang, Su-Ping Chen, Yang Zhang, Qing Liu, Liang Zhou, Xiao-Gang Jiang, Kifayat Ullah, Qing Guo, Zhao-Hui Liu, Ying Xu, Jun-Hai Han, Jiajie Hou, Darran P O'Connor, Guoqiang Xu

引用次数: 0

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出版商更正：E3泛素连接酶UBR5通过促进关键时钟转录因子BMAL1的泛素化和降解来调节昼夜节律。

本文章由计算机程序翻译，如有差异，请以英文原文为准。

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来源期刊

Acta Pharmacologica Sinica 医学-化学综合

CiteScore

15.10

自引率

2.40%

发文量

4365

审稿时长

2 months

期刊介绍： APS (Acta Pharmacologica Sinica) welcomes submissions from diverse areas of pharmacology and the life sciences. While we encourage contributions across a broad spectrum, topics of particular interest include, but are not limited to: anticancer pharmacology, cardiovascular and pulmonary pharmacology, clinical pharmacology, drug discovery, gastrointestinal and hepatic pharmacology, genitourinary, renal, and endocrine pharmacology, immunopharmacology and inflammation, molecular and cellular pharmacology, neuropharmacology, pharmaceutics, and pharmacokinetics. Join us in sharing your research and insights in pharmacology and the life sciences.