Retraction: “Quercetin improve ischemia/reperfusion-induced cardiomyocyte apoptosis in vitro and in vivo study via SIRT1/PGC-1α signaling”

IF 3 3区生物学 Q3 BIOCHEMISTRY & MOLECULAR BIOLOGY

Journal of cellular biochemistry Pub Date : 2024-04-03 DOI:10.1002/jcb.30554

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引用次数: 0

Abstract

Retraction: “Quercetin improve ischemia/reperfusion-induced cardiomyocyte apoptosis in vitro and in vivo study via SIRT1/PGC-1α signaling”, by Jiayou Tang, Linhe Lu, Yang Liu, Jipeng Ma, Lifang Yang, Lanlan Li, Hong Guo, Shiqiang Yu, Jun Ren, Heping Bai, Jian Yang, J Cell Biochem. 2019; 120: 9747-9757: The above article, published online on 17 January 2019 in Wiley Online Library (https://onlinelibrary.wiley.com/doi/full/10.1002/jcb.28255) has been retracted by agreement between the journal's Editor in Chief, Christian Behl, and Wiley Periodicals LLC.

The retraction has been agreed following an investigation based on allegations raised by a third party. Several flaws and inconsistencies between results presented and experimental methods described were found. Furthermore, image elements in Figure 4 were found to have been previously published elsewhere in a different scientific context. Thus, the editors consider the conclusions of this article to be invalid. The authors did not respond when asked to collaborate during the investigation and confirm the retraction.

查看原文本刊更多论文

撤稿："槲皮素通过SIRT1/PGC-1α信号转导改善缺血/再灌注诱导的心肌细胞凋亡的体外和体内研究"

撤稿："槲皮素通过SIRT1/PGC-1α信号转导改善缺血/再灌注诱导的心肌细胞凋亡的体外和体内研究》，作者：唐家友、卢林和、刘洋、马继鹏、杨丽芳、李兰兰、郭红、于世强、任军、白和平、杨健，《细胞生物化学杂志》。2019; 120: 9747-9757：2019年1月17日在线发表在《Wiley Online Library》（https://onlinelibrary.wiley.com/doi/full/10.1002/jcb.28255）上的上述文章已被该杂志主编Christian Behl和Wiley Periodicals LLC协议撤回。根据第三方提出的指控进行调查后，双方同意撤回文章。在对第三方提出的指控进行调查后，该期刊同意撤回论文。调查发现，论文所提供的结果与所描述的实验方法之间存在若干缺陷和不一致之处。此外，还发现图 4 中的图像元素以前曾在其他不同的科学背景下发表过。因此，编辑认为这篇文章的结论无效。在被要求合作调查并确认撤稿时，作者没有做出回应。

本文章由计算机程序翻译，如有差异，请以英文原文为准。

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来源期刊

Journal of cellular biochemistry 生物-生化与分子生物学

CiteScore

9.90

自引率

0.00%

发文量

164

审稿时长

1 months

期刊介绍： The Journal of Cellular Biochemistry publishes descriptions of original research in which complex cellular, pathogenic, clinical, or animal model systems are studied by biochemical, molecular, genetic, epigenetic or quantitative ultrastructural approaches. Submission of papers reporting genomic, proteomic, bioinformatics and systems biology approaches to identify and characterize parameters of biological control in a cellular context are encouraged. The areas covered include, but are not restricted to, conditions, agents, regulatory networks, or differentiation states that influence structure, cell cycle & growth control, structure-function relationships.