[Progression and stagnation of periodontal disease].

Stomatologie der DDR Pub Date : 1990-05-01
K Sally
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引用次数: 0

Abstract

Local factors may contribute to acute exacerbations of the periodontal disease, however, their pathological basis is systemically established. The more bacteria and bacterial products get into the periodontal tissue the more serious is destruction. Host's resistance controls composition of the subepithelial bacterial flora. Under condition of defective phagocyte function bacteria, otherwise occurring rarely in the pocket, multiply quickly and penetrate the periodontal pocket epithelium. Specific immune defence can stop them only in the subepithelial conjunctive tissue causing its destruction. Juvenile periodontitis goes with defects of granulocyte function and an aggressive pocket flora. These patients need antibiotics taken systematically, removing of the infected pocket epithelium and regular recalls. Care of patients with adult periodontitis is limited to good oral hygiene.

[牙周病的进展和停滞]。
局部因素可能导致牙周病的急性加重,但其病理基础是系统建立的。进入牙周组织的细菌和细菌产物越多,对牙周组织的破坏就越严重。宿主的抵抗力控制着上皮下细菌菌群的组成。在吞噬细胞功能缺陷的情况下,原本在牙周袋中很少出现的细菌迅速繁殖并渗透到牙周袋上皮中。特异性免疫防御只能在上皮下结缔组织中阻止它们,导致其破坏。青少年牙周炎伴有粒细胞功能缺陷和侵袭性口袋菌群。这些患者需要系统地服用抗生素,去除感染的口袋上皮并定期召回。成人牙周炎患者的护理仅限于良好的口腔卫生。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
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