Atherosclerosis: Causes and Cures

S. Evangeline, P. Iyer
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引用次数: 1

Abstract

Atherosclerosis is a systemic disease that occurs due to the formation of Fibro-fatty lesions in arteries that block blood flow. The process of Atherosclerosis is initiated through mechanical, chemical, and immunological activation of the endothelium. Even there are microorganisms involved in the different stages of atherosclerosis. There are a number of molecular factors, RNA’s, macrophages and enzymes that play a vital role in the progression and development of Atherosclerosis that alter the metabolism of Fibro-fatty. Receptors of micro RNA proliferate and regulate endothelial activation and smooth cell Macrophages, monocytes, t-cell and natural killer cells involved in the boosting of the immune system to prevent the atherosclerotic lesion formation. In this review, we can know how RNA’s involved in the pathophysiology side of atherosclerosis and explore the mechanism to regulate Atherosclerosis, and how macrophages evoke an immune response. Therefore, the use of synthetic and natural drugs and following the right diet timely prevent and reduce the risk of development of Atherosclerosis.
动脉粥样硬化:原因和治疗
动脉粥样硬化是一种全身性疾病,由于动脉中纤维脂肪病变的形成阻碍了血液流动而发生。动脉粥样硬化的过程是通过内皮细胞的机械、化学和免疫激活而开始的。甚至在动脉粥样硬化的不同阶段也有微生物参与。有许多分子因子,RNA,巨噬细胞和酶在动脉粥样硬化的进展和发展中起着至关重要的作用,改变了纤维脂肪的代谢。微RNA受体增殖并调节内皮细胞活化和平滑细胞巨噬细胞、单核细胞、t细胞和自然杀伤细胞参与增强免疫系统,防止动脉粥样硬化病变的形成。通过本文,我们可以了解RNA如何参与动脉粥样硬化的病理生理方面,并探讨其调节动脉粥样硬化的机制,以及巨噬细胞如何引起免疫反应。因此,使用合成药物和天然药物,并遵循正确的饮食,及时预防和降低动脉粥样硬化的发生风险。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
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