Transmitting, mutational and pandemic nature of corona: the role of transposons, is1: tn:is1 and crispr- cas-9

Abstract

Corona/ Covid19/Omicron / SARS/ MARS similar viruses have no individual genetic mechanism to make them encapsulated spore forming microbes similar to bacteria, fungi, algal blooms involved spreading allergy. Transmission in all such cases need fluid dynamics of water and air. This flow does not require any support of matrix, i.e., nano particles of carbon, silicates and dust of animate inanimate molecules. They enter into lung alveoli surface epithelial cell, adhere on the surface of body saliva, originated due to cold and other infections. In support of ACE-I and II (Angiotensin Converting Enzyme I and II). So, there are two releasing mechanisms are existing in corona infection; one is the release of corona virus from the alveoli cells with matrix binds of one co-morbid patients directly to other healthy recipient at the distance of 6 m in a closed environment by its self-development irritation and allergy, as received by sudden irritating smoke developed from kitchen, pollen, that grows during grass cutting, cleaning and sanitization. Transcription of m-RNA, in a matrix based single corona after entering into lung alveoli starts to transcribe and translate to many molecules of corona viruses, damages alveoli cells of lung, caused due to individual physiological conditions, old (above 70 years) and young (below 40), co-morbid(sick) and healthy people at similar age groups. IS: Tn:IS), Crispr/Cas-9 DNA transposons existing in corona m-RNA starts to act during transcription, and translation