Increased Expression of Oxidative Stress in Proliferated Vascular Smooth Muscle Cells in Early Stage of Human Restenosis After Coronary Angioplasty

Abstract

The repair of vascular injury is a concerted response involving complex signaling pathway that contributes to the clinical feature of post─angioplasty restenosis . Vascular injury leads to the activation of various biological cells and the release of numerous vasoactive, thrombogenic, and mitogenic factors in the process of restenosis. Restenosis is histologically attributed to neointimal hyperplasia Original Articles