S. Sánchez-Ovando, S. Pavlidis, N. Kermani, K. Baines, D. Barker, P. Gibson, L. Wood, I. Adcock, K. Chung, J. Simpson, P. Wark
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引用次数: 2
Abstract
Severe asthma (SA) is a heterogeneous disease. Transcriptomic analysis contributes to the understanding of pathogenesis necessary for developing new therapies. We sought to identify and validate mechanistic pathways of SA across two independent cohorts.
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